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Updated: Jan 7, 2026

Isolation of Mouse Respiratory Epithelial Cells and Exposure to Experimental Cigarette Smoke at Air Liquid Interface
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Isolation of Mouse Respiratory Epithelial Cells and Exposure to Experimental Cigarette Smoke at Air Liquid Interface

Published on: February 21, 2011

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NR8383細胞とCOPDのラットモデルにおけるmiR-122/PTEN/AKT軸の調節によるタバコ煙によるアポトーシス

Yang Jiang1, Xiaosheng Jin2

  • 1Department of Science and Education, Tongde Hospital of Zhejiang Province, Hangzhou, 310012, Zhejiang, P.R.China.

Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals
|January 3, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
COPD についてPTEN についてネズミの煙のモデルアポトーシスmiR-122 について

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Chronic Obstructive Pulmonary Disease (COPD) pathophysiology is intricate and multifaceted, involving a complex interplay of physiological processes. Understanding these mechanisms is crucial for effectively managing and treating COPD. Here is an in-depth look at the critical elements in the pathophysiology of COPD:
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Chronic obstructive pulmonary disease (COPD) is a group of lung conditions that progressively worsen over time, including chronic bronchitis and emphysema. This cluster of diseases collectively leads to a gradual and irreversible decline in lung function over time.
The primary cause for the onset of COPD is cigarette smoking and exposure to air pollution. These hazardous factors initiate a chain reaction within the lungs, resulting in chronic inflammation, damage to the airways, and a...
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