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タンシノーンVIは,マウスの心不全の進行を弱める

Tetsuro Marunouchi1, Yuji Kawahara2, Kouichi Tanonaka1

  • 1Department of Molecular and Cellular Pharmacology, Tokyo University of Pharmacy and Life Sciences.

Biological & pharmaceutical bulletin
|January 4, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
心不全と保存されたエジェクション分数 (HFpEF)ミトゲン活性化タンパク質キナーゼタンシノンVI

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The activation of the sympathetic nervous system and the renin-angiotensin-aldosterone system (RAAS) contributes to cardiac remodeling, and inhibiting the RAAS is a pharmacological target in heart failure management. As a result, neurohumoral modulation is a crucial treatment principle for managing heart failure. This approach involves using medications like ACE inhibitors (ACEIs), angiotensin receptor blockers (ARBs), β-blockers, mineralocorticoid receptor antagonists (MRAs), and neutral...
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