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Updated: Jan 7, 2026

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ミトコンドリアとシナプス経路を交差させ,ALSの病理性を改善したGM1

Federica Pilotto1,2, Tristan Dellazizzo Toth3,4, Silvano Bond3,4

  • 1Institut Neuromyogène, Pathophysiology and genetics of the neuron and muscle, Inserm U1315, CNRS, Université Claude Bernard Lyon I, Lyon, France.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|January 5, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
C9ORF72 についてERとミトコンドリアの接触GM1 についてSOD1‐G93Aについてアミオトロフィック横筋硬化症 (ALS)ミトコンドリア機能障害ナノリポソーマル製剤プロテオミクスシナプス機能障害

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