関連する実験動画
Updated: Feb 13, 2026

04:24
Chuzhen Therapy as a Non-Invasive Traditional Chinese Therapy for Neck Pain
Published on: June 6, 2025
1.3K
乳がんに対する内分泌療法に関する中国の専門家の一致 (2026年版)
Yang Yuan1, Yongmei Yin2, Shu Wang3
1Senior Department of Oncology, Chinese People's Liberation Army (PLA) General Hospital, Beijing, China.
まとめ
更新された2026年のコンセンサスは,ホルモン受容体陽性乳がんに対する内分泌療法 (ET) に関するガイドラインを提供し,早期および進行段階の新しいデータを統合しています. ネオアジュバントETの適格性と,アジュバントサイクリン依存キナーゼ4/6阻害剤 (CDK4/6i) の使用を明確にしています.
科学分野:
- 腫瘍学 腫瘍学
- エンドクリノロジー エンドクリノロジー
- クリニカル・プラクティスのガイドライン
背景:
- 内分泌療法 (ET) は,ホルモン受容体 (HR) 陽性乳がんの基礎治療である.
- 乳がん内分泌療法に関する中国コンセンサスグループは,最近の臨床研究と実務経験に基づいて2023年のコンセンサスを更新しました.
研究 の 目的:
- HR陽性乳がんの標準化および個別化治療のための最新の臨床ガイドラインを提供すること.
- 最新の研究データと専門家の経験を臨床実践の勧告に統合する.
主な方法:
- 医学腫瘍学者,乳房外科医,病理学者を含む専門家グループが設立されました.
- 2025年9月30日までの主要な英語データベースで,包括的な文献検索が行われました.
- 証拠の質と勧告の強さは,中国臨床腫瘍学会 (CSCO) のガイドラインフレームワークを使用して評価されました.
主要な成果:
- 2026年のコンセンサスには,HR陽性乳がんの初期および進行時の最新データが含まれています.
- それは,ネオアジュバントETの適格性を定義し,アジュバントのサイクリン依存キナーゼ4/6阻害剤 (CDK4/6i) 療法の使用を明確にします.
- 先進的な疾患およびCDK4/6i後のレジミンの第一線ETとCDK4/6iに対する最適化された治療戦略は,遺伝子検査に関するガイドラインとともに推奨されます.
結論:
- この更新されたコンセンサスは,ETの標準化された適用のための臨床ガイドラインを提供します.
- それは,HR陽性乳がん患者のための正確で個別化された治療戦略を促進することを目的としています.
関連する概念動画
Endocrine Signaling
68.2K
Endocrine cells produce hormones to communicate with remote target cells found in other organs. The hormone reaches these distant areas using the circulatory system. This exposes the whole organism to the hormone but only those cells expressing hormone receptors or target cells are affected. Thus, endocrine signaling induces slow responses from its target cells but these effects also last longer.
68.2K
Cancer Therapies
10.2K
Cancer therapies are various modes of treatment, such as surgery, radiation therapy, and chemotherapy that are administered to cancer patients.
However, cancer treatments can pose several challenges, as therapies used to kill cancer cells are generally also toxic to normal cells. Moreover, cancer cells mutate rapidly and can develop resistance to chemical agents or radiation therapy. Besides, all types of cancer cells may not respond to the same therapy. Some cancer cells respond to one...
However, cancer treatments can pose several challenges, as therapies used to kill cancer cells are generally also toxic to normal cells. Moreover, cancer cells mutate rapidly and can develop resistance to chemical agents or radiation therapy. Besides, all types of cancer cells may not respond to the same therapy. Some cancer cells respond to one...
10.2K
Targeted Cancer Therapies
9.0K
The targeted cancer therapies, also known as “molecular targeted therapies,” take advantage of the molecular and genetic differences between the cancer cells and the normal cells. It needs a thorough understanding of the cancer cells to develop drugs that can target specific molecular aspects that drive the growth, progression, and spread of cancer cells without affecting the growth and survival of other normal cells in the body.
There are several types of targeted therapies against...
There are several types of targeted therapies against...
9.0K
What is the Endocrine System?
421.4K
The endocrine system sends hormones—chemical signals—through the bloodstream to target cells—the cells the hormones selectively affect. These signals are produced in endocrine cells, secreted into the extracellular fluid, and then diffuse into the blood. Eventually, they diffuse out of the blood and bind to target cells which have specialized receptors to recognize the hormones.
421.4K
The Endocrine System
1.4K
The endocrine system is an extensive network of glands – organs or tissues in the body that create chemicals that control many bodily functions, that secrete hormones, which are chemical messengers that play essential roles in regulating various bodily functions. These hormones are secreted into the bloodstream and travel throughout the body. They require specific receptors to convey signals to cells possessing these corresponding receptors. This complex signaling mechanism ensures that...
1.4K
RNA Editing
9.9K
RNA editing is a post-transcriptional modification where a precursor mRNA (pre-mRNA) nucleotide sequence is changed by base insertion, deletion, or modification. The extent of RNA editing varies from a few hundred bases, in mitochondrial DNA of trypanosomes, to a just single base, in nuclear genes of mammals. Even a single base change in the pre-mRNA can convert a codon for one amino acid into the codon for another amino acid or a stop codon. This type of re-coding can significantly affect the...
9.9K

