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Updated: Feb 14, 2026

Incorporating Pericytes into an Endothelial Cell Bead Sprouting Assay
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FOXF2は,新生児の高酸素性肺損傷後の血管ホメオスタシスに必要なペリサイト・エンドセリアルシグナリングを調節する

Fei Sun1,2,3, Yuchen Zhao4, Jonathan Do5

  • 1Key Laboratory of Birth Defects and Related Diseases of Women and Children of MOE, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, China. feisun@scu.edu.cn.

Nature communications
|February 12, 2026
PubMed
まとめ

FOXF2は,肺血管の発達におけるペリサイト機能に不可欠です. FOXF2の喪失は血管形成とアルベオライゼーションを阻害し, bronchopulmonary dysplasiaに寄与する.

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