妊娠中の母親の栄養不良は,子羊の筋肉形態測定,コラーゲン遺伝子発現,および羊の肉の繊細さを変化させます
Mia Y Kawaida1, Amanda S Reiter1, Nicole M Tillquist1
1Department of Animal Science, University of Connecticut, Storrs, CT 06269, USA.
Animals : an open access journal from MDPI
|February 13, 2026
まとめ
妊娠中の母親の食事は,子孫の肉の品質に影響します. 制限されたまたは過度の給餌により,筋肉繊維のサイズと繊細性が変化し,羊肉のコラーゲン含有量と遺伝子発現に影響を与えます.
科学分野:
- 動物科学 動物科学
- ミートサイエンスの科学
- 発達生物学 発達生物学について
背景:
- 妊娠中の母親の栄養は胎児の発達に不可欠です.
- 栄養学的なプログラミングは,子孫の成長,代謝,および組織特性に影響を与えることができます.
- 肉の品質特性に母親の食事の影響を理解することは,畜産業界にとって不可欠です.
研究 の 目的:
- 妊娠中の母親の過度な給餌と制限された給餌が,コラーゲン含有量,筋肉繊維の横断面 (CSA),および子孫の肉の繊細さに及ぼす影響を調査する.
- 筋肉の発達と羊の筋肉のコラーゲン合成に関連する遺伝子発現に対する母親の食事の影響を分析する.
主な方法:
- 妊娠中の雌羊は,妊娠中にコントロール (100%),制限 (60%) または過剰 (140%) の食事を割り当てられました.
- 男性の子孫は,約282日目の年齢で評価されました.
- 筋肉CSA,ワーナー・ブラッツラー剪定力 (WBSF),遺伝子発現 (ミオジェニックおよびフィブロジェニック) は,ロングシマス (LM) とセミテンディノス (STN) 筋肉で定量化されました.
主要な成果:
- 制限され,過剰に餌を与えられた子孫は,対照群と比較して,半筋CSAが小さかった.
- 過剰に餌を与えられた子孫は,ロングシシマス筋CSAの減少を示し,制限された子孫は,ロングシシマスCSAの拡大を示した.
- 過剰に餌を与えられた子孫は,ロングシマス筋肉のWBSFが減少したことを示しました.
- 母親の食生活は,LMとSTNの両方の筋肉における重要なミオジェニックおよびファイブロジェニック遺伝子の発現に影響を与えました.
結論:
- 妊娠中の母親の栄養状態は,子孫の筋肉の発達と肉の品質の特徴を大幅に変化させます.
- 妊娠中の特定の食事の介入は,筋肉繊維のサイズ,肉の柔らかさ,およびコラーゲン合成経路に影響を与える可能性があります.
- これらの発見は,高品質の肉製品を生産するために最適な母性栄養の重要性を強調しています.
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