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Updated: Feb 15, 2026

Activation and Measurement of NLRP3 Inflammasome Activity Using IL-1β in Human Monocyte-derived Dendritic Cells
09:04

Activation and Measurement of NLRP3 Inflammasome Activity Using IL-1β in Human Monocyte-derived Dendritic Cells

Published on: May 22, 2014

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炭素系ナノマテリアルは,グランウラサ細胞におけるNLRP3炎症ゾーム媒介のピロプトーシス経由で卵巣機能障害を誘発する

Simin Wei1, Dan Chen1, Jingxin Wang1

  • 1Department of Obstetrics and Gynecology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China; National Clinical Research Center for Obstetrical and Gynecological Diseases, Wuhan, Hubei 430030, China; Key Laboratory of Cancer Invasion and Metastasis, Ministry of Education, Wuhan, Hubei 430030, China.

Journal of advanced research
|February 13, 2026
PubMed
まとめ

炭素ベースのナノマテリアルは,NLRP3炎症体活性化による花粉素細胞のピロプトーシスを引き起こすことによって卵巣機能を破壊します. NLRP3を阻害することは,ナノマテリアルによって引き起こされる卵巣損傷に対する潜在的な治療法を提供します.

キーワード:
炭素ベースのナノマテリアルグランウロサ細胞は,グランウロサ細胞である.NLRP3は炎症性である.卵巣の機能についてピロプトーシス (Pyroptosis) というものです.繁殖性毒性 繁殖性毒性について

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