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2つのCCM2分子のKRIT1への二重徴募は,KLF4発現を抑制する

Clotilde Huet-Calderwood1, Oriana S Fisher1,2, Sreya Das1

  • 1Department of Pharmacology, Yale University, New Haven, CT, USA.

Nature communications
|February 13, 2026
PubMed
まとめ

2つのCCM2タンパク質が1つのKRIT1に集まって,KLF4発現を調節し,脳血管疾患である脳洞性異常を予防しなければなりません. これは,内皮細胞の維持に不可欠な新しいPTBドメインの募集メカニズムを明らかにします.

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