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Updated: Feb 16, 2026

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COPDは,NSCLCの腫瘍マイクロ環境を再構成し,抗PD-1治療への応答を強化します

Xiaoke Chen1, Fang Ye2, Sikun Liu1

  • 1Department of Thoracic Surgery, Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, China.

Med (New York, N.Y.)
|February 14, 2026
PubMed
まとめ

慢性阻害性肺疾患 (COPD) は,腫瘍の微小環境を改造することによって,PD-1ブロックに対する非小細胞肺がん (NSCLC) の反応を高めます. これは,マクロファージを勧誘するCXCL14-CXCR4軸が関与し,T細胞の浸透を促進し,免疫療法の結果を改善します.

キーワード:
COPD (慢性肺炎症) は,慢性肺炎 (COPD) と呼ばれる疾患で,慢性肺炎 (COPD) は慢性肺炎 (COPD) と呼ばれる疾患で,慢性肺炎 (COPD) は慢性肺炎 (COPD) と呼ばれる疾患で,慢性肺炎 (COPD) は慢性肺炎 (COPD) と呼ばれる疾患で,慢性肺炎 (COPD) は慢性肺炎 (COPD) と呼ばれる疾患で,慢性肺炎 (COPD) は慢性肺炎 (COPD) と呼ばれています.CXCL14はCXCL14と一致している.NSCLCLCは,NSCLCLCは,NSCLCLCは,NSCLCLCは,NSCLCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは,NSCLCは抗PD-1治療法による治療である.免疫療法の有効性について患者さんへの翻訳腫瘍のマイクロ環境

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