シグモイド板の脱音が,パルサチル型耳鳴りのために,横側鼻腔ステントを施した後に再発した
Piers Klein1, Pamela Semaan2, Thanh N Nguyen1
1Department of Neurology, Radiology, Boston University Chobanian & Avedisian School of Medicine, Boston, Massachusetts, USA.
Journal of neurointerventional surgery
|February 14, 2026
まとめ
シグモイドプレート脱光症 (SPD) の骨の再生は,脈動性耳鳴 (PT) の内血管治療後に一般的であった. これは,SPDが得られたものであり,手術を必要としない可能性があることを示唆しています.
科学分野:
- 神経外科は神経外科です.
- 血管外科 血管外科
- 放射線学 放射線学
背景:
- シグモイドプレート脱音症 (SPD) は,脈動性耳鳴 (PT) の患者で頻繁に観察されます.
- SPDの正確な原因は議論されているが,横鼻狭窄症 (TSS) による血液動力学的変化がますます関与している.
- SPDはシグモイド板の欠如を伴い,シグモイドシヌスとマストイド空気細胞の間の直接的な接触を可能にします.
研究 の 目的:
- 脈動性耳鳴 (PT) の血管内治療後のシグモイド板消音 (SPD) の変化を評価する.
- 治療後のシグモイド板骨の再生率と特徴を決定する.
主な方法:
- PTに対する静脈内静脈治療を受けている15人の患者の遡及的なコホート研究.
- 脱光症は,マストイド空気の細胞と直接のシナスの接触を示したCT画像で定義された.
- 2人の神経放射線科医は,治療前のおよび後のCTスキャンを使用して,骨の再生を評価しました.
主要な成果:
- シグモイド板の骨の再生は,治療後の患者の86.7%で発生しました.
- 再生は,主として血管内干渉 (92.3%) に対してipsilateralであった.
- 臨床的または手続的要因に関して,再成長した患者と再成長していない患者の間で有意な違いが見つかりませんでした.
結論:
- ほとんどの患者はPTの内血管治療後にシグモイド板骨の再生を経験しました.
- これらの発見は,SPDはおそらく既得症状であることを示唆しています.
- SPDは,治療後の骨の再生が自発的に起こる可能性があるため,外科的介入を必要としない場合があります.
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