関連する実験動画

Updated: Jun 12, 2026

Use of a Hanging-weight System for Liver Ischemia in Mice
05:53

Use of a Hanging-weight System for Liver Ischemia in Mice

Published on: August 7, 2012

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DRP1の枯渇はNK細胞を低酸素誘発機能障害から保護する

Tias Verhezen1, Astrid Van Den Eynde1, Peter Verstraelen2

  • 1Center for Oncological Research (CORE), Integrated Precision and Personalized Oncology Network (IPPON), University of Antwerp, Antwerpen, Belgium.

Redox report : communications in free radical research
|February 18, 2026
PubMed
まとめ

低酸素症は,固体腫瘍における自然殺傷細胞 (NK細胞) の機能を損なう. DRP1タンパク質を非活性化すると,NK細胞のミトコンドリアと細胞毒性の活性が低毒状態で回復し,CAR-NK細胞の有効性を高めます.

キーワード:
CRISPR-Cas9は,CRISPR-Cas9と一致している.がん がん がん がん がんDRP1 DRP1は,DRP1を表示している.低酸素症とはミトコンドリアはミトコンドリアです.自然のキラー細胞は,ROSOS ROSは,ROSOSという名前で呼ばれています.腫瘍のマイクロ環境

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The Unfolded Protein Response01:37

The Unfolded Protein Response

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Export of Misfolded Proteins out of the ER01:32

Export of Misfolded Proteins out of the ER

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