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Updated: May 2, 2026

Stereological and Flow Cytometry Characterization of Leukocyte Subpopulations in Models of Transient or Permanent Cerebral Ischemia
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Stereological and Flow Cytometry Characterization of Leukocyte Subpopulations in Models of Transient or Permanent Cerebral Ischemia

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オゾンによって引き起こされる認知欠陥は,肝-脳軸によって媒介される:外周補足体C3がマイクログリアルシナプスファゴシトーシスを誘発する

Yougang Wang1,2, Haomin Qi1,2, Weiran Dong1

  • 1Hubei Provincial Clinical Research Center for Alzheimer's Disease, Brain Science and Advanced Technology Institute, School of Medicine, Wuhan University of Science and Technology, Wuhan, 430081, China.

Journal of neuroinflammation
|February 20, 2026
PubMed
まとめ

オゾン (O3) 曝露は,脳のシナプスを損傷することによって認知を損なう. これは,オゾンがコンプリメントコンポーネント3 (C3) の放出を誘発し,マイクログリアの活性化とシナプスの喪失につながる肝臓-脳軸を通じて起こります.

キーワード:
オゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンオゾンコグニティブ障害 (Cognitive Impairment) とは,認知機能の低下によるコンプリメントC3はC3です.肝臓 脳の軸マイクログリアはミクログリア.

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