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Updated: Feb 24, 2026

Balloon-based Injury to Induce Myointimal Hyperplasia in the Mouse Abdominal Aorta
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マウスにおける血管内膜肥厚を駆動する血管平滑筋特異的NLRP3過剰活性化

Yun-Ting Wang, Alexandra K Moura, Rui Zuo

    Research square
    |February 23, 2026
    PubMed
    まとめ

    血管平滑筋細胞（VSMC）特異的NLRP3インフラマソームの過剰活性化は、内膜肥厚および再狭 stenosis を駆動します。VSMC NLRP3-TFEBシグナル伝達を標的とすることは、血管疾患の進行およびプラークの蓄積を制限する可能性があります。

    キーワード:
    血管平滑筋細胞NLRP3インフラマソーム内膜肥厚再狭 stenosisオートファジーTFEB血管疾患

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