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Updated: Jul 7, 2026

Transfer of Manipulated Tumor-associated Neutrophils into Tumor-Bearing Mice to Study their Angiogenic Potential In Vivo
08:19

Transfer of Manipulated Tumor-associated Neutrophils into Tumor-Bearing Mice to Study their Angiogenic Potential In Vivo

Published on: July 20, 2019

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慢性腫瘍壊死因子-アルファ阻害薬（TNF-I）曝露と泌尿生殖器悪性腫瘍のリスク：多施設共同後ろ向きコホート研究

Conor B Driscoll1, Jordan M Rich2, Christopher Yang3

  • 1Department of Urology, Northwestern University Feinberg School of Medicine, 676 N. St. Clair Street, Arkes 23, Chicago, IL, 60611, USA. conor.driscoll@northwestern.edu.

World journal of urology
|February 26, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
炎症前立腺がん腫瘍壊死因子アルファ阻害薬泌尿生殖器がん

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Tumor Necrosis Factor (TNF), a proinflammatory cytokine, contributes significantly to the inflammation seen in Crohn's disease. It exists as soluble TNF and membrane-bound TNF, with actions mediated through TNF receptors (TNFR). TNFR activation leads to the release of proinflammatory cytokines, T-cell activation, collagen production, and leukocyte migration, all contributing to inflammation in Crohn's disease. Anti-TNF monoclonal antibodies, namely infliximab (Remicade), adalimumab (Humira),...