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Updated: Feb 28, 2026

Assessment of Mitochondrial Functions and Cell Viability in Renal Cells Overexpressing Protein Kinase C Isozymes
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DUSP26はセリン312でのp53の脱リン酸化により急性腎障害を防御する

Ying Fu1,2,3, Yu Xiang1, Yun Han1

  • 1Department of Nephrology, Hunan Key Laboratory of Kidney Disease and Blood Purification, Institute of Nephrology, The Second Xiangya Hospital at Central South University, Changsha, China.

Nature communications
|February 26, 2026
PubMed

キーワード:
急性腎障害DUSP26p53腎尿細管障害治療標的

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