アデノウイルスは,コールド・スプリング・ハーバーで驚かされる.

Same author

NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.

Mucosal immunology·2017

Same author

The impact on utilities of differences in body weight among Canadian patients with type 2 diabetes.

Current medical research and opinion·2014

Same author

Mammalian inositol polyphosphate 5-phosphatase II can compensate for the absence of all three yeast Sac1-like-domain-containing 5-phosphatases.

The Biochemical journal·2001

Same author

The yeast inositol polyphosphate 5-phosphatases inp52p and inp53p translocate to actin patches following hyperosmotic stress: mechanism for regulating phosphatidylinositol 4,5-bisphosphate at plasma membrane invaginations.

Molecular and cellular biology·2000

Same author

Binding sites for Hsp70 molecular chaperones in natural proteins.

Cold Spring Harbor symposia on quantitative biology·1995

Same author

The cellular response to unfolded proteins: intercompartmental signaling.

Current opinion in biotechnology·1994

関連する実験動画

アデノウイルスは,コールド・スプリング・ハーバーで驚かされる.

関連する概念動画

NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.

The impact on utilities of differences in body weight among Canadian patients with type 2 diabetes.

Mammalian inositol polyphosphate 5-phosphatase II can compensate for the absence of all three yeast Sac1-like-domain-containing 5-phosphatases.

The yeast inositol polyphosphate 5-phosphatases inp52p and inp53p translocate to actin patches following hyperosmotic stress: mechanism for regulating phosphatidylinositol 4,5-bisphosphate at plasma membrane invaginations.

Binding sites for Hsp70 molecular chaperones in natural proteins.

The cellular response to unfolded proteins: intercompartmental signaling.

関連する実験動画

アデノウイルスは,コールド・スプリング・ハーバーで驚かされる.

関連する概念動画

NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.

The impact on utilities of differences in body weight among Canadian patients with type 2 diabetes.

Mammalian inositol polyphosphate 5-phosphatase II can compensate for the absence of all three yeast Sac1-like-domain-containing 5-phosphatases.

The yeast inositol polyphosphate 5-phosphatases inp52p and inp53p translocate to actin patches following hyperosmotic stress: mechanism for regulating phosphatidylinositol 4,5-bisphosphate at plasma membrane invaginations.

Binding sites for Hsp70 molecular chaperones in natural proteins.

The cellular response to unfolded proteins: intercompartmental signaling.

関連する実験動画

アデノウイルスは,コールド・スプリング・ハーバーで驚かされる.

関連する概念動画

関連記事

NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.

The impact on utilities of differences in body weight among Canadian patients with type 2 diabetes.

Mammalian inositol polyphosphate 5-phosphatase II can compensate for the absence of all three yeast Sac1-like-domain-containing 5-phosphatases.

The yeast inositol polyphosphate 5-phosphatases inp52p and inp53p translocate to actin patches following hyperosmotic stress: mechanism for regulating phosphatidylinositol 4,5-bisphosphate at plasma membrane invaginations.

Binding sites for Hsp70 molecular chaperones in natural proteins.

The cellular response to unfolded proteins: intercompartmental signaling.

関連する実験動画

アデノウイルスは,コールド・スプリング・ハーバーで驚かされる.

関連する概念動画

関連記事

NOX1 loss-of-function genetic variants in patients with inflammatory bowel disease.

The impact on utilities of differences in body weight among Canadian patients with type 2 diabetes.

Mammalian inositol polyphosphate 5-phosphatase II can compensate for the absence of all three yeast Sac1-like-domain-containing 5-phosphatases.

The yeast inositol polyphosphate 5-phosphatases inp52p and inp53p translocate to actin patches following hyperosmotic stress: mechanism for regulating phosphatidylinositol 4,5-bisphosphate at plasma membrane invaginations.

Binding sites for Hsp70 molecular chaperones in natural proteins.

The cellular response to unfolded proteins: intercompartmental signaling.