D A Kaku1, R G Giffard, D W Choi

  • 1Department of Neurology and Neurological Sciences, Stanford University School of Medicine, CA 94305.

Science (New York, N.Y.)
|June 4, 1993
PubMed
まとめ

細胞外酸性は,ニューロンを酸素欠乏から保護し,グルタミン酸対抗剤の神経保護効果を高めます. この組み合わせは,長期にわたる低酸素状態下でも,重要なニューロン生存率を提供します.

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