A Krook1, M A Soos, S Kumar

  • 1Department of Medicine, University of Cambridge, Addenbrooke's Hospital, UK.

PubMed
まとめ

新しいモノクローナル抗体は,ラブソン-メンデンホール症候群における変異性インスリン受容体 (Ser323Leu) を活性化させ,インスリン結合の欠陥を回避し,潜在的な治療用途のためのインスリンシグナル伝達経路を復元することができます.

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