ERの品質管理: 細胞プラズマの接続

R R Kopito¹

¹Department of Biological Sciences, Stanford University, California 94305-5020, USA.

Cell

|February 21, 1997

まとめ

No abstract available in PubMed .

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関連する実験動画

ERの品質管理: 細胞プラズマの接続

関連する概念動画

Loss of polyubiquitin gene Ubb leads to metabolic and sleep abnormalities in mice.

Effect of ubiquitin expression on neuropathogenesis in a mouse model of familial amyotrophic lateral sclerosis.

Specificity in intracellular protein aggregation and inclusion body formation.

Impairment of the ubiquitin-proteasome system by protein aggregation.

Aggresomes and Russell bodies. Symptoms of cellular indigestion?

Aggresomes, inclusion bodies and protein aggregation.

A viral ORFeome library for systems-level genetic dissection of host-pathogen interactions.

Co-option of lysosomal machinery shapes the evolution of the intracellular photosymbiosis supporting coral reefs.

LEF1 and niche factors determine T cell stemness across chronic diseases.

Recurrent patterns of TOP1-mediated neuronal genomic damage shared by major neurodegenerative disorders.

Four-dimensional molecular mapping from a spatial snapshot reveals the dynamics of hair follicle organogenesis.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

関連する実験動画

ERの品質管理: 細胞プラズマの接続

関連する概念動画

Loss of polyubiquitin gene Ubb leads to metabolic and sleep abnormalities in mice.

Effect of ubiquitin expression on neuropathogenesis in a mouse model of familial amyotrophic lateral sclerosis.

Specificity in intracellular protein aggregation and inclusion body formation.

Impairment of the ubiquitin-proteasome system by protein aggregation.

Aggresomes and Russell bodies. Symptoms of cellular indigestion?

Aggresomes, inclusion bodies and protein aggregation.

A viral ORFeome library for systems-level genetic dissection of host-pathogen interactions.

Co-option of lysosomal machinery shapes the evolution of the intracellular photosymbiosis supporting coral reefs.

LEF1 and niche factors determine T cell stemness across chronic diseases.

Recurrent patterns of TOP1-mediated neuronal genomic damage shared by major neurodegenerative disorders.

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関連する実験動画

ERの品質管理: 細胞プラズマの接続

関連する概念動画

関連記事

Loss of polyubiquitin gene Ubb leads to metabolic and sleep abnormalities in mice.

Effect of ubiquitin expression on neuropathogenesis in a mouse model of familial amyotrophic lateral sclerosis.

Specificity in intracellular protein aggregation and inclusion body formation.

Impairment of the ubiquitin-proteasome system by protein aggregation.

Aggresomes and Russell bodies. Symptoms of cellular indigestion?

Aggresomes, inclusion bodies and protein aggregation.

A viral ORFeome library for systems-level genetic dissection of host-pathogen interactions.

Co-option of lysosomal machinery shapes the evolution of the intracellular photosymbiosis supporting coral reefs.

LEF1 and niche factors determine T cell stemness across chronic diseases.

Recurrent patterns of TOP1-mediated neuronal genomic damage shared by major neurodegenerative disorders.

Four-dimensional molecular mapping from a spatial snapshot reveals the dynamics of hair follicle organogenesis.

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関連する実験動画

ERの品質管理: 細胞プラズマの接続

関連する概念動画

関連記事

Loss of polyubiquitin gene Ubb leads to metabolic and sleep abnormalities in mice.

Effect of ubiquitin expression on neuropathogenesis in a mouse model of familial amyotrophic lateral sclerosis.

Specificity in intracellular protein aggregation and inclusion body formation.

Impairment of the ubiquitin-proteasome system by protein aggregation.

Aggresomes and Russell bodies. Symptoms of cellular indigestion?

Aggresomes, inclusion bodies and protein aggregation.

A viral ORFeome library for systems-level genetic dissection of host-pathogen interactions.

Co-option of lysosomal machinery shapes the evolution of the intracellular photosymbiosis supporting coral reefs.

LEF1 and niche factors determine T cell stemness across chronic diseases.

Recurrent patterns of TOP1-mediated neuronal genomic damage shared by major neurodegenerative disorders.

Four-dimensional molecular mapping from a spatial snapshot reveals the dynamics of hair follicle organogenesis.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.