相关实验视频
Updated: Jul 15, 2026

10:10
Quantifying Cognitive Decrements Caused by Cranial Radiotherapy
Published on: October 18, 2011
手持蜂电话的使用和脑癌的风险
J E Muscat1, M G Malkin, S Thompson
1American Health Foundation, 1 Dana Rd, Valhalla, NY 10595, USA. jmuscat2@earthlink.net
JAMA
|December 21, 2000
概括
使用手机似乎不会增加脑癌风险. 需要进一步的研究来证实这些发现,特别是缓慢生长的瘤.
科学领域:
- 神经科学是一个神经科学.
- 流行病学 流行病学
- 在瘤学瘤学.
背景情况:
- 关于手机使用对健康的影响的数据有限.
- 调查手机使用与健康结果之间的潜在联系至关重要.
研究的目的:
- 调查手持手机使用与原发性脑癌风险之间的关联.
- 测试使用手机可能是脑瘤的危险因素的假设.
主要方法:
- 从1994年到1998年,在美国5个学术医疗中心进行了一项病例控制研究.
- 469名原发性脑癌患者和422名匹配对照被采访,使用结构化问卷.
- 通过比较病例和对照之间的手机使用时间和使用年数来评估风险.
主要成果:
- 定期使用手持手机和脑癌风险之间没有发现显著的关联 (OR,0.85;95% CI,0.6-1.2).
- 与非使用者相比,不经常 (<0.72小时/月) 和频繁 (>10.1小时/月) 使用者都没有增加风险.
- 使用时间与脑癌风险没有相关性 (P =.54).
- 一个轻微的,不显著的趋势表明瘤发生在与使用手机相同的头部侧面 (P = .06),但这并不一致于所有瘤类型.
结论:
- 目前的数据表明,手持蜂电话的使用与原发性脑癌的风险增加无关.
- 较长的随访时间和瘤潜伏期的考虑是全面了解的必要条件,特别是缓慢生长的瘤.
相关概念视频
Biological Effects of Radiation
All radioactive nuclides emit high-energy particles or electromagnetic waves. When this radiation encounters living cells, it can cause heating, break chemical bonds, or ionize molecules. The most serious biological damage results when these radioactive emissions fragment or ionize molecules. For example, α and β particles emitted from nuclear decay reactions possess much higher energies than ordinary chemical bond energies. When these particles strike and penetrate matter, they produce ions...
Cancers Originate from Somatic Mutations in a Single Cell
Cancer arises from mutations in the critical genes that allow healthy cells to escape cell cycle regulation and acquire the ability to proliferate indefinitely. Though originating from a single mutation event in one of the originator cells, cancer progresses when the mutant cell lines continue to gain more and more mutations, and finally, become malignant. For example, chronic myelogenous leukemia (CML) develops initially as a non-lethal increase in white blood cells, which progressively...
Mouse Models of Cancer Study
Mice have long served as models for studying human biology and pathology because of their phylogenetic and physiological similarity with humans. They are also easy to maintain and breed in the laboratory, and hence, many inbred strains are now available for research. Studies on mice have contributed immeasurably to our understanding of cancer biology.
The development of transgenic, knockout, and knock-in mice has led to an exponential increase in their use as model organisms in research,...
The development of transgenic, knockout, and knock-in mice has led to an exponential increase in their use as model organisms in research,...
Cancer Prevention
Several factors can increase the risk of cancer in an individual. About 50% of cancer cases can be prevented by adopting a healthy lifestyle, regular exercise, eating healthy, and following a modest cancer prevention diet. Epidemiological studies have consistently shown that populations with vegetable and fruit-rich diets have reduced the incidence of cancer. On the other hand, populations who have a diet rich in animal fat, red meat, junk food, or high calories are predisposed to cancer.
Some...
Some...
Cancers Originate from Somatic Mutations in a Single Cell
Cancer arises from mutations in the critical genes that allow healthy cells to escape cell cycle regulation and acquire the ability to proliferate indefinitely. Though originating from a single mutation event in one of the originator cells, cancer progresses when the mutant cell lines continue to gain more and more mutations, and finally, become malignant. For example, chronic myelogenous leukemia (CML) develops initially as a non-lethal increase in white blood cells, which progressively...
Mutagenicity and Carcinogenicity
Mutagenicity and carcinogenicity refer to the ability of drugs to cause genetic defects and induce cancer, respectively. The International Agency for Research on Cancer (IARC) classifies agents into four groups based on their carcinogenic potential. Group 1 agents are known human carcinogens; group 2A agents are probably carcinogenic to humans; group 3 agents lack data to support their role in carcinogenesis; and group 4 includes agents for which data support that they are not likely to be...

