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通过皮肤冠状动脉干预后离散的微发作可视化,与轻微的肌酸激酶-MB升高相关

M J Ricciardi¹, E Wu, C J Davidson

¹Feinberg Cardiovascular Institute and the Department of Medicine, Northwestern University, Chicago, IL, USA.

|June 13, 2001

概括

穿皮冠状动脉干预后肌酸激酶-MB (CK-MB) 轻度升高表明微发作. 与对比度增强的MRI可视化了这种心肌损伤,与CK-MB水平相关.

科学领域:

心脏病学心脏病学
医疗成像医学成像

背景情况:

在皮肤冠状动脉干预 (PCI) 后,轻微的肌酸激酶-MB (CK-MB) 升高是常见的.
在PCI后释放CK-MB背后的机制尚未完全理解.
与对比度增强的MRI提供了心肌缩的直接可视化.

研究的目的:

研究PCI后轻度CK-MB升高的机制.
为了将心肌损伤的生物化学标记与使用MRI的解剖学发现相关联.
评估对比度增强型MRI在检测手术相关心肌损伤方面的实用性.

主要方法:

没有先前心脏病发作的14名患者在PCI后接受了cine和对比度增强的MRI.
患者根据CK-MB升高 (9有,5没有) 进行了分类.
评估MRI超增强和墙壁运动异常;测量CK-MB水平.

主要成果:

所有9名CK-MB升高的患者在MRI上都显示出离散的超增强,表明肌.
肌病的估计质量中位数为2.0g (1.5%的LV质量).
只有一个患者出现了新的墙壁运动异常;在大多数重复扫描中,超增强仍然存在.

结论:

与对比度增强的MRI证实了与手术相关的心肌损伤,尽管心电图或壁面运动正常.
在PCI后,轻微的CK-MB升高是离散微发作的结果.
磁力共振成像为心肌损伤的生化证据提供了解剖学的基础.

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