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I Shiraishi¹, T Takamatsu, T Minamikawa

¹Department of Pathology, Children's Research Hospital, Kyoto, Japan.

|June 1, 1992

概括

鼻腔 (SA) 节点生长到成年,SA节点细胞的体积增加. 衰老导致SA结节细胞缩和脂肪透,而不仅仅是连接组织的替换.

科学领域:

心血管研究研究心血管研究
历史学历史学历史学
生物老龄化生物老龄化

背景情况:

纤维化和鼻腔 (SA) 节点的脂肪透通常被解释为由结缔组织替代SA节点细胞.
以前的解释缺乏精确的定量分析来验证组织学变化.

研究的目的:

在人类发育和衰老过程中对SA结节细胞和结缔组织的体积变化进行定量分析.
为了澄清SA节点卷入的组织学基础.

主要方法:

利用数字彩色图像分析仪计算从串行截面的SA节点细胞和结缔组织的体积.
采用细胞光测量来确定SA结节细胞的DNA ploidy 模式.

主要成果:

从婴儿期到成年期,SA结节细胞体积增加了2.4倍,而SA结节总体体积增加了4.2倍,这表明连接组织显著扩张 (7.4倍).
在老年人中,SA节点和SA节点细胞体积下降,但纤维结合组织体积保持不变.
在生长过程中,SA结节细胞不会合成DNA,如常数DNA ploidy所示.

结论:

在成年之前明显的SA结节细胞的卷积是一种错误的印象,这是由于间歇性组织的比例较高而引起的.
随着年龄的增长,SA结节细胞缩发生,伴随着SA结节的减少和/或脂肪透.

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