编程细胞死亡和Vbeta8+ CD4+ T 细胞的异常减少,在对黄金葡萄球菌 (Staphylococcus aureus) 肠毒素B耐受性小鼠中

Y Kawabe1, A Ochi

  • 1Department of Immunology and Medical Genetics, University of Toronto, Ontario, Canada.

Nature
|January 17, 1991
PubMed
概括

通过克隆缺失可以诱导外围T细胞耐受性. 研究表明,在小鼠中暴露于金黄色葡萄球菌肠毒素B导致特定T细胞的减少,表明细胞死亡有助于耐受性.