腺A2A受体激活对心肌梗塞的缓解作用是由于它对CD4+ T淋巴细胞的作用

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Zequan Yang1, Yuan-Ji Day, Marie-Claire Toufektsian

  • 1Department of Biomedical Engineering, University of Virginia Health System, Box 800759, Charlottesville, VA 22903, USA. zy6b@virginia.edu

Circulation
|October 25, 2006
PubMed
概括

氨酸A2A受体 (A2AR) 的激活通过抑制CD4+T细胞来减少心脏病发作损伤. 这项研究揭示了CD4+ T淋巴细胞是肌肉心脏缺血症/再输液损伤的关键贡献者.

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