快照:细胞体的细胞体

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Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

bioRxiv : the preprint server for biology·2026

Same author

A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Cellular and molecular life sciences : CMLS·2026

Same author

Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Molecular therapy. Oncology·2025

Same author

Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

Nature communications·2025

Same author

On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

bioRxiv : the preprint server for biology·2025

Same author

LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Molecular cancer research : MCR·2025

Same journal

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Cell·2026

Same journal

Systematic discovery of pathogen effector functions across human pathogens and pathways.

Cell·2026

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Structural basis for host membrane binding and remodeling by invading malaria parasites.

Cell·2026

Same journal

Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

Cell·2026

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Arc mediates intercellular tau transmission via extracellular vesicles.

Cell·2026

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Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.

Cell·2026

相关实验视频

快照:细胞体的细胞体

相关概念视频

Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Systematic discovery of pathogen effector functions across human pathogens and pathways.

Structural basis for host membrane binding and remodeling by invading malaria parasites.

Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

Arc mediates intercellular tau transmission via extracellular vesicles.

Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.

相关实验视频

快照:细胞体的细胞体

相关概念视频

相关文章

Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Systematic discovery of pathogen effector functions across human pathogens and pathways.

Structural basis for host membrane binding and remodeling by invading malaria parasites.

Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

Arc mediates intercellular tau transmission via extracellular vesicles.

Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.