使

E M Schuman1, D V Madison

  • 1Department of Molecular and Cellular Physiology, Beckman Center for Molecular and Genetic Medicine, Stanford University School of Medicine, CA 94305-5426.

Science (New York, N.Y.)
|December 6, 1991
PubMed
概括

氧化 (NO) 可能在长期强化 (LTP) 中充当逆行信使. 抑制NO合成或结合NO阻断LTP,表明NO从后突触神经元释放对于前突触LTP表达至关重要.