K2P1/TWIK1K+?

Sylvain Feliciangeli1, Saïd Bendahhou, Guillaume Sandoz

  • 1Institut de Pharmacologie Moléculaire et Cellulaire, CNRS UMR6097, Institut Paul Hamel, 660, route des lucioles, 06560 Valbonne, France.

Cell
|August 19, 2007
PubMed
概括

一种新型模型表明,细胞兴奋性是由K2P1通道化调节的. 突变特定部位 (K274E) 增加了K2P1的电流,表明充电效应,而不是化.

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