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Updated: May 10, 2026

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瘤抑制剂Par-4激活了细胞亡的外部途径

Ravshan Burikhanov1, Yanming Zhao, Anindya Goswami

  • 1Department of Radiation Medicine, University of Kentucky, Lexington, KY 40536, USA.

Cell
|July 28, 2009
PubMed
概括

前列腺亡反应-4 (Par-4) 是由细胞分泌的,并通过结合细胞表面GRP78.8诱导癌细胞亡. 这种细胞外Par-4信号激活了死亡途径,提供了一个潜在的治疗目标.

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