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对具有减弱细胞致病性VSV突变的分析:病毒功能的突变,P,用于抑制蛋白质合成

C P Stanners, A M Francoeur, T Lam

    Cell
    |June 1, 1977
    PubMed
    概括

    T1026是一种囊泡性口炎病毒 (VSV) 突变,由于双重突变,其致病性降低,并导致持续感染. 这种双重突变影响RNA聚合酶和病毒蛋白合成,为持续性病毒感染研究提供了一个模型.

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