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在唐氏综合征的Tc1小鼠模型中,瘤血管生成减少

Louise E Reynolds1, Alan R Watson, Marianne Baker

  • 1Adhesion and Angiogenesis Laboratory, Barts Institute of Cancer, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, John Vane Science Centre, Charterhouse Square, London EC1M 6BQ, UK. l.reynolds@qmul.ac.uk

Nature
|June 11, 2010
PubMed
概括

具有额外副本的人类染色体21基因的小鼠显示瘤生长和血管生成减少. 这是由于特定的基因抑制了血管内皮生长因子 (VEGF) 反应,提供了潜在的癌症标.

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Mice have long served as models for studying human biology and pathology because of their phylogenetic and physiological similarity with humans. They are also easy to maintain and breed in the laboratory, and hence, many inbred strains are now available for research. Studies on mice have contributed immeasurably to our understanding of cancer biology.
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