流产性艾滋病毒感染中介于人类淋巴细胞组织的CD4 T细胞枯竭和炎症

Gilad Doitsh1, Marielle Cavrois, Kara G Lassen

  • 1Gladstone Institute of Virology and Immunology, San Francisco, CA 94158, USA.

Cell
|November 30, 2010
PubMed
概括

艾滋病毒通过流产性感染消耗CD4 T细胞,引发宿主防御反应. 这种机制涉及不完整的病毒遗传物质,导致被编程的细胞死亡和炎症,有助于艾滋病毒的发病.

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