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氧化作为发展重塑过程中轴突退化和再生之间的切换机制

Dana Rabinovich1, Shiri P Yaniv1, Idan Alyagor1

  • 1Department of Molecular Cell Biology, Weizmann Institute of Sciences, Rehovot 7610001, Israel.

Cell
|January 16, 2016
PubMed
概括

神经元在成长和削减状态之间切换. 通过E75核受体调节的氧化 (NO) 信号控制这种开关,促进切割,同时抑制神经元发育过程中的再生.

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