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碳氧甲基纤维素作为食品乳化剂:它的日子已经数了吗?
Eduardo M Costa1, Sara Silva1, Carla F Pereira1
1CBQF-Centro de Biotecnologia e Química Fina-Laboratório Associado, Escola Superior de Biotecnologia, Universidade Católica Portuguesa, Rua Diogo Botelho 1327, 4169-005 Porto, Portugal.
碳氧甲基纤维素 (CMC) 可能会促进肠道炎症,尽管它常见的使用和公认的安全性. 这项研究发现,CMC在肠道上皮细胞中诱导了前炎性反应,这表明对其未来纳入食品的考虑.
科学领域:
- 胃肠病学 胃肠病学
- 免疫学 免疫学 免疫学
- 食品科学 食品科学 食品科学
背景情况:
- 碳氧甲基纤维素 (CMC) 在工业中广泛使用,并被监管机构普遍认为是安全的.
- 新兴研究表明存在潜在的安全问题,包括与CMC相关的肠道失生症.
- 胃肠道中CMC的促炎潜力仍然没有得到解决.
研究的目的:
- 为了调查Carboxymethyl纤维素 (CMC) 是否作为一种促进炎症的化合物.
- 评估CMC对胃肠道上皮细胞的免疫调节作用.
主要方法:
- 对暴露于CMC的Caco-2,HT29-MTX和Hep G2细胞进行了细胞毒性测试.
- 在Caco-2细胞单层中测量了促炎性细胞因子分泌 (IL-6,IL-8,TNF-α,IL-1β).
- 涉及Caco-2细胞和RAW 264.7巨细胞的共同培养模型被用于评估细胞因子概况.
主要成果:
- 在高达25毫克/毫升的度下,CMC对测试细胞系没有细胞毒性.
- 在Caco-2细胞中,CMC显著增加了IL-6,IL-8和TNF-α的分泌,TNF-α显著增加了1924%.
- 共同培养模型揭示了复杂的细胞因子反应,包括促炎和抗炎标志物.
结论:
- 碳氧甲基纤维素 (CMC) 在肠道光膜中表现出一种促炎性行为.
- 需要进一步的研究才能充分了解CMC对肠道健康的影响.
- 将CMC纳入食品需要仔细考虑,以减轻潜在的胃肠道失调症.
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