激活Pgk1导致在各种神经退行性疾病中减少蛋白质聚合

Hao Chen1, Yajie Li1, Jingwen Gao1

  • 1Department of Biochemistry and Molecular Biology School of Basic Medicine, Capital Medical University, Youanmen, Beijing, 100069, China.

PubMed
概括

现在 (TZ) 通过激活糖酸酶1 (Pgk1) 和Hsp90.0. 通过激活神经退行性疾病模型中预防病态蛋白质凝聚物. 这种候选药物增加了ATP水平并促进了自,提供了潜在的治疗策略.