介绍神经可塑性叙述:一种非病理化的生物基础,用于创伤信息和不良童年经历意识的方法
1Centre for Mental Health Nursing, School of Health Sciences, University of Melbourne, Carlton, VIC, Australia.
Frontiers in psychiatry
|June 7, 2023
概括
童年不良经历 (ACE) 和创伤通过神经可塑性塑造了我们的生物学,影响了心理痛苦. 神经可塑性叙述为这种痛苦提供了一个非病理化的进化解释,指导创伤知情护理.
科学领域:
- 神经科学是一个神经科学.
- 心理学 心理学 心理学
- 进化生物学 进化生物学
背景情况:
- 大多数在心理健康服务中的个人报告了不良童年经历 (ACE) 和/或复杂的创伤史.
- 目前以创伤为基础的方法缺乏对创伤和后来的痛苦之间的联系的生物学解释.
- 这种差距导致痛苦被误诊并被视为精神疾病.
研究的目的:
- 介绍神经可塑性叙述,一个神经生态学理论.
- 提供一个生物框架,将创伤和逆境与心理痛苦联系起来.
- 为创伤知情和ACEs意识的心理健康护理提供一个非病理化的基础.
主要方法:
- 阐述神经可塑性叙事理论.
- 整合神经可塑性的概念 (表观遗传学,神经发生,突触可塑性,白质可塑性).
- 将理论置于进化框架内,补充生命史和附着理论.
主要成果:
- 情绪和心理上的痛苦被概念化为在不利的环境中生存的进化成本.
- 神经可塑性机制将生活经验嵌入到生物学中,创建适应性 (良性) 或不适应性 (恶性) 循环.
- 该理论认为,痛苦源于适应,而不是病理,因为大脑的健康功能是适应.
结论:
- 神经塑性叙述为理解与创伤和ACE相关的痛苦提供了生物学基础.
- 将与创伤相关的痛苦误诊为精神疾病可能会导致阳性损害并使耻辱永久化.
- 这种进化,非病理化的框架支持心理健康中的创伤知情和ACEs意识方法.
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