分子机制调解罗夫米拉斯的保护作用,防止异上腺素诱导的心肌损伤

Marwa Monier Mahmoud Refaie1, Manar Fouli Gaber Ibrahim2, Michael Atef Fawzy3

  • 1Department of Pharmacology, Faculty of Medicine, Minia University, El-Minia, Egypt.

概括

罗夫米拉斯 (ROF) 通过调节PDE4和关键信号通路,保护免受异上腺素 (ISO) 诱导的心肌损伤. 这种治疗证明了抗氧化,抗炎和抗丧作用,为心脏损伤提供了潜在的治疗策略.

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