Acinetobacter baumannii的外膜蛋白A通过TLR2-NF-κB通路调节肺炎

Yumei Li1, Laibing Liu2, Chaolun Xiao1

  • 1Department of Anatomy, School of Basic Medical Sciences, Guizhou Medical University/ Department of Nephrology, Guiyang First People's Hospital, Guiyang, Guizhou 550025, China.

PubMed
概括

菌毒性因子OmpA通过TLR2-NF-κB通路激活NLRP3炎症体,增强炎症. 这一途径是治疗耐药A. baumannii感染的潜在目标.

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