氧化应激和COVID-19相关的神经元功能障碍:机制和治疗影响
Dylan R Bowen1, Suhrud Pathak1, Rishi M Nadar1
1Department of Drug Discovery and Development, Harrison College of Pharmacy, Auburn University, Auburn-AL 36849, USA.
Acta biochimica et biophysica Sinica
|June 26, 2023
概括
氧化压力可能在COVID-19患者的神经问题中发挥关键作用. 需要进一步的研究来理解这些机制,并开发治疗COVID-19相关的神经并发症的方法.
科学领域:
- 神经科学是一个神经科学.
- 传染性疾病 传染性疾病
- 生物化学 生物化学
背景情况:
- 严重急性呼吸系统综合征 (SARS) -CoV-2 导致COVID-19,新出现的证据将其与神经功能障碍联系起来.
- 氧化应激越来越被认为是各种疾病病理生理学的潜在贡献者,包括神经系统疾病.
结论:
- 氧化压力与COVID-19引起的神经功能障碍的发病有关.
- 需要进一步的机械和临床研究来充分阐明这些影响.
- 准氧化应激途径可能为缓解COVID-19的神经并发症提供治疗潜力.
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