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Updated: Jul 25, 2025

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P300增加了CSNK2A1的表达,通过激活PI3K-AKT-mTOR轴加速结直肠癌的进展

Jilong Liu1

  • 1Tumor Surgical Department, Beijing Chuiyangliu Hospital, No.2, Chuiyangliu South Street, Chaoyang District, Beijing, 100022, China.

Experimental cell research
|June 30, 2023
PubMed
概括

这项研究表明,P300增强了素激酶2α1 (CSNK2A1) 的表达,通过激活PI3K-AKT-mTOR通路来促进结直肠癌 (CRC) 的进展. 沉默CSNK2A1可以抑制CRC细胞的迁移和入侵.

关键词:
在CSNK2A1A1中.结肠直肠癌是一种癌症.H3K27ac 在线阅读这是入侵的入侵.移民 移民 移民在P300300中,P300是最重要的.这就是PI3K-AKT-mTOR.

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