视觉神经压缩导致的ipsilateral视觉损失,这种视觉损失发生在结核细胞脑膜瘤和内 Carotid 动脉之间的视觉神经压缩
Ryunosuke Hashikawa1, Noriaki Fukuhara1, Mitsuo Yamaguchi-Okada1
1Department of Hypothalamic and Pituitary Neurosurgery, Toranomon Hospital, Toranomon, Minato, Japan.
Asian journal of neurosurgery
|July 3, 2023
概括
一个罕见的视力丧失病例是由于肺结核 sellae 脑膜瘤 (TSM) 和内动脉 (ICA) 压缩视神经而发生的. 这发生在没有瘤侵入视通道的情况下,突出显示了一种非典型的呈现.
科学领域:
- 神经外科 神经外科
- 眼科医生 眼科 眼科
- 神经学 神经学
背景情况:
- 结核 sellae 脑膜瘤 (TSMs) 可以通过压缩视神经引起视力障碍.
- 视通道干扰是TSM相关视力损失的常见因素.
- 需要仔细考虑TSM的非典型表现.
研究的目的:
- 报告一种罕见的视觉神经压缩引起的双侧视力损失病例.
- 描述一种不寻常的压缩机制,涉及内动脉 (ICA).
- 强调在TSM情况下考虑非透性压缩的重要性.
主要方法:
- 评估了一名70岁的女性患者,她有2年的视力障碍病史.
- 磁共振成像 (MRI) 被用于诊断TSM.
- 进行了扩展的内镜转形手术以切除瘤.
主要成果:
- 术前成像显示,瘤没有透到视通道.
- 完成了TSM的手术切除.
- 在TSM和动脉样硬化性内动脉 (ICA) 之间确定了视神经压缩.
结论:
- 双侧视力损失可能是由于TSM和ICA对视神经的压缩造成的,即使没有视通道透.
- 这一案例突出了TSM引起的视力障碍的非典型机制.
- 内镜手术是有效的TSM去除和解压视神经.
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