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通过不活跃的β-arrestin对E3泛素酶MDM2的GPCR向

Yaejin Yun1, Hye-Jin Yoon1, Yejin Jeong2

  • 1Department of Chemistry, College of Natural Sciences, Seoul National University, Seoul 08826, Republic of Korea.

Proceedings of the National Academy of Sciences of the United States of America
|July 3, 2023
PubMed
概括

E3 泛素酶Mdm2 结合于非活性β-arrestin1,促进G蛋白结合受体 (GPCR) 的内部化. 这种相互作用对于受体信号调节至关重要.

关键词:
G蛋白结合受体的受体.在被逮捕后被捕.在MDM2中,MDM2是MDM2.在任何地方都是无处不在的.

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