[由于牙过载而导致的铜缺乏的椎骨髓病:一个病例报告]
M Polo Royo1, B B Hidalgo Mendía1, R Jariod Gaudes2
1F.E.A. Unidad de Lesionados Medulares, Hospital Miguel Servet, Zaragoza, España.
Rehabilitacion
|July 3, 2023
概括
假牙中的中毒导致铜缺乏,导致骨髓病变. 用铜和提取即时治疗逆转了神经损伤,改善了功能.
科学领域:
- 神经学 神经学
- 毒理学 毒理学 毒理学
- 营养科学 营养科学
背景情况:
- 亚急性骨髓病症可能伴有感觉和运动缺陷.
- 中毒是一种潜在的,虽然不常见的,导致铜缺乏的原因.
研究的目的:
- 报告一种中毒后的骨髓病的病例.
- 突出铜缺乏症的诊断和治疗影响.
主要方法:
- 一个68岁的老人病例介绍,他的神经症状逐渐恶化.
- 磁共振成像 (MRI) 用于评估脊髓干涉.
- 血液检查以确定铜和的含量.
- 用铜补充治疗和去除含的假牙粘合剂.
- 康复包括物理治疗,水疗和职业治疗.
主要成果:
- 这位患者出现了手臂和胸部低听觉和步行动力衰竭,这表明后髓病变.
- 铜缺乏症的诊断是在假牙粘剂中中毒的次要结果.
- 治疗导致功能改善,ASIAD评分从C4变为C7.
- 神经学缺陷被扭转,防止不可逆转的损伤.
结论:
- 在不压缩性骨髓病与后干涉的情况下,应检查铜缺乏症.
- 早期诊断和干预,包括补充铜和撤销,对于恢复至关重要.
- 这一案例凸显了考虑营养缺乏的催生性原因的重要性.
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