桑托胡摩尔通过通过AMPK/Akt/GSK3β通路的Nrf2激活来保护对乙氨基诱导的肝毒性

Laiyu Zhu1, Xiaoye Fan1, Chunyuan Cao2

  • 1Institute of Translational Medicine, The First Hospital of Jilin University, Changchun, Jilin 130001, China.

概括

香醇通过减少氧化应激和线粒体损伤来保护肝脏免受乙氨基过量. 它激活Nrf2通路,为乙胺诱导的肝毒性提供了潜在的治疗策略.

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