通过调节TGF-β1/Smad3通路介导的自,SMOC2在心力衰竭中发挥作用

Yu Ren1, Yun Wu2,3, Wenshuai He2,3

  • 1Scientific Research Department, Inner Mongolia People's Hospital, Inner Mongolia Autonomous Region, Hohhot, 010017, China.

PubMed
概括

分泌的蛋白质酸性和丰富的囊相关的模块化结合蛋白2 (SMOC2) 通过影响自和TGF-β1 / Smad3通路促进心力衰竭 (HF). 减少SMOC2在高频模型中改善了心脏功能.

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