CBD:对抗脱发,脱发和其潜在机制的潜在头
1Department of Pharmacy, Pranveer Singh Institute of Technology (Pharmacy) NH19 Bhaunti Kanpur, India.
Current drug discovery technologies
|July 21, 2023
概括
大麻素 (CBD) 通过与身体的内分泌大麻素系统 (ECS) 相互作用,对治疗脱发和脱发有希望. 需要进一步的临床研究来证实它在修复头发方面的有效性和安全性.
科学领域:
- 皮肤病学和三叶病学
- 药理学 药理学是指药理学的学科.
- 自然产品研究自然产品研究
背景情况:
- 脱发影响了相当一部分人口,导致心理困扰.
- 现有的脱发治疗方法通常具有有限的疗效和潜在的副作用.
- 大麻素 (CBD) 正在成为治疗脱发的潜在治疗剂,因为它与内分泌系统 (ECS) 相互作用.
研究的目的:
- 通过CBD进行深入审查脱发及其管理策略.
- 阐明CBD影响头发生长并对抗脱发症的作用机制.
- 巩固目前对CBD在治疗脱发中的作用的科学理解.
主要方法:
- 在包括PubMed,Google Scholar,Scopus,ScienceDirect,Wiley和Springer在内的主要科学数据库中进行全面的文献搜索.
- 对研究的分析,重点关注CBD在治疗脱发和脱发症中的应用,疗效和机制.
- 对植物性大麻素及其潜在治疗效果的研究进行审查.
主要成果:
- 基于CBD的疗法为治疗脱发和脱发症提供了一种新的方法.
- 详细介绍了各种类型的脱发及其常规治疗方法.
- 该审查强调了CBD作用的潜在机制,包括其与ECS内的CB1和CB2受体的相互作用.
结论:
- 现有的文献支持使用CBD治疗脱发症的科学基础.
- 局部和口服CBD的使用显示了治疗脱发的潜力.
- 需要进一步的临床前和临床研究来确定CBD在头发修复中的有效性和安全性.
相关概念视频
Accessory Structures of the Skin: Hair Growth and Types
1.5K
Hair growth begins with the production of keratinocytes by the basal cells of the hair bulb. As new cells are deposited at the hair bulb, the hair shaft is pushed through the follicle toward the surface. Keratinization is completed as the cells are pushed to the skin surface to form the shaft of hair that is externally visible. The external hair is completely dead and composed entirely of keratin. Hair can be cut or shaven without damaging the hair structure because the cut is superficial. Most...
1.5K
CNS Stimulants: Cocaine, Amphetamines and Cannabinoids
242
CNS stimulants, such as cocaine, amphetamines, and cannabinoids, have varying structures and mechanisms of action that lead to different therapeutic effects and side effects. Cocaine, with its molecular formula C17H21NO4, is a tropane alkaloid and a tertiary amino compound. It has two chemical forms: the hydrochloride salt and the "freebase." The former is in powder form, while the latter involves removing the hydrochloride salt to create a form that can be smoked. Cocaine exerts its...
242
Alzheimer's Disease: Treatment
219
Alzheimer's Disease (AD), a neurodegenerative disorder, is pathologically identified by amyloid plaques and neurofibrillary tangles composed of tau protein. AD pharmacotherapy aims to manage cognitive symptoms, delay disease progression, and treat behavioral symptoms. The treatment is primarily symptomatic and palliative, with no definitive disease-modifying therapy available. Cholinesterase inhibitors, including donepezil (Aricept), rivastigmine (Exelon), and galantamine (Razadyne), are...
219
Multipotency and Niche of Bulge Stem Cell
3.7K
A hair follicle or HF is a small part of the skin that produces the hair shaft. Paul Gerson Unna was the first to observe a bulge in the human hair follicle's outer root sheath (ORS). The bulge is present between the sebaceous gland and the arrector pili muscle and is the niche for hair follicle stem cells (HFSCs). The bulge is also a niche for melanocyte stem cells, and their loss results in graying of hair. The HFSCs express Sox9 and Lhx2, which help them maintain stemness and prevent...
3.7K
Chemotherapy-Induced Nausea and Vomiting: Cannabinoids
322
Tetrahydrocannabinol (THC) is a phytocannabinoid that primarily interacts with the CB1 receptor, a type of G protein-coupled receptor (GPCR) predominantly in and around the chemoreceptor trigger zone (CTZ) and emetic center. THC also blocks the serotonin receptor activity in the dorsal vagal complex (DVC) by inhibiting serotonin release. THC exerts its anti-emetic effects through these interactions, which are beneficial for patients undergoing chemotherapy.
Two synthetic agonists of THC,...
Two synthetic agonists of THC,...
322
Alzheimer's Disease: Overview
525
Alzheimer's Disease (AD) is a continually advancing neurodegenerative disorder, distinguished by escalating memory loss, cognitive dysfunction, and dementia. The disease unfolds in three stages: preclinical, mild cognitive impairment (MCI), and dementia. Its onset is insidious, and the progression gradual, with the cause not well explained by other disorders.
The clinical diagnosis of AD hinges on the presence of memory and other cognitive impairments. Biomarkers, such as changes in Aβ...
The clinical diagnosis of AD hinges on the presence of memory and other cognitive impairments. Biomarkers, such as changes in Aβ...
525


