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Updated: Jul 20, 2026

In ovo Electroporation of miRNA-based Plasmids in the Developing Neural Tube and Assessment of Phenotypes by DiI Injection in Open-book Preparations
12:41

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通过向SDF1来调节神经细胞-码细胞相互作用的乙醇诱导的干扰,外体转运的miR-126通过向SDF1来调节

Yihong Li1,2,3,4, Ting Cai1,2, Huina Liu1,2

  • 1Ningbo No.2 Hospital, Ningbo 315099, China.

Toxicological sciences : an official journal of the Society of Toxicology
|July 25, 2023
PubMed
概括

乙醇通过破坏神经细胞 (NCC) 和斑点细胞 (PC) 之间的相互作用来破坏胎儿发育. 从暴露于乙醇的NCC中携带miR-126的外体抑制PC中的SDF1,导致胎儿酒精谱系障碍 (FASD) 的面缺陷.

关键词:
这就是SDF1.外基因组是外基因组的组成部分.在 miR-12626 系统中,神经顶细胞是神经顶细胞.斑点细胞 斑点细胞

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