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多态性是否预测功能性腹痛障碍儿童的催眠疗法反应:一项探索性研究

Clara M A de Bruijn1, Stefan W Hovy2, Ellen Tromp3

  • 1From the Emma Children's Hospital, Amsterdam UMC, University of Amsterdam, Pediatric Gastroenterology, Hepatology and Nutrition, Amsterdam, The Netherlands.

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在COMT,OPRM1和MAO-A中的遗传变异不能预测功能性腹痛障碍儿童的催眠疗法反应. 这项研究发现,这些遗传多态化与儿科患者的治疗成功之间没有关联.

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科学领域:

  • 遗传学 是一个遗传学.
  • 儿科 儿科 儿科
  • 心理学 心理学 心理学

背景情况:

  • 特定的基因多态性 (COMT,OPRM1,MAO-A) 与成年人的催眠能力有关.
  • 催眠疗法 (HT) 在儿童的功能性腹痛障碍 (FAPD) 中显示出有效性.
  • 在儿科FAPD中HT反应的遗传基础仍然未被探索.

研究的目的:

  • 研究COMT,OPRM1和MAO-A基因多态化与FAPD儿童的催眠治疗成功之间的关联.
  • 探索这些多形态与缓解疼痛,焦虑和生活质量等二次结果之间的潜在联系.

主要方法:

  • 探索性研究涉及144名儿科患者 (8-18岁) 患有FAPD,来自先前的HT试验.
  • 采集的口腔样本用于COMT,OPRM1和MAO-A多态的基因定型.
  • 分析基因型与3个月HT后治疗成功之间的关联,以及二次结果措施.

主要成果:

  • 在儿科FAPD患者中,COMT,MAO-A和OPRM1多态变异和治疗成功 (TS) 之间没有发现显著的关联.
  • 这些遗传多态性并不能预测足够的缓解,焦虑,抑郁,生活质量,人体化,催眠易感性,期望,疼痛信念或应对策略.

结论:

  • COMT,OPRM1和MAO-A基因多态性似乎不是诊断有功能性腹痛障碍的儿科患者催眠治疗反应的预测因素.
  • 可能需要进一步的研究来确定影响这群人群中催眠治疗结果的遗传或其他因素.