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在商业肉中对细菌性冠状炎和骨髓炎的病理生理学和分子机制的审查

Venkata Sesha Reddy Choppa¹, Woo Kyun Kim¹

¹Department of Poultry Science, University of Georgia, Athens, GA 30602, USA.

|July 29, 2023

概括

现代肉由于快速生长,容易患有细菌性冠状炎和骨髓炎 (BCO). 益生菌,益生菌和协生菌可以通过促进肠道健康和减少细菌转移来帮助预防BCO.

科学领域:

兽医病理学兽医病理学
微生物学微生物学
动物科学动物科学

背景情况:

现代肉的生长速度很快,导致新陈代谢需求和对机会性病原体的易感性.
细菌性冠状炎和骨髓炎 (BCO) 源于病原体在运动器官中的定居,由微骨折和缺血加剧.
骨质稳定被包括急性阶段反应和细胞死亡途径在内的因素破坏,并非所有受影响的鸟类都表现出临床.

研究的目的:

审查BCO.的潜在和新生物标志物.
探索与BCO相关的病理生理机制,经济意义和免疫反应.
研究肠道微生物组和细胞培养模型在了解BCO.CO.中的作用.

主要方法:

文献综述侧重于BCO病原体,风险因素和缓解策略.
细菌从肠道疾病转移到骨感染的分析.
探索益生菌,益生菌和共生菌对BCO发病率的影响.

主要成果:

鉴定了BCO.中涉及的常见和不可培养的细菌病原体.
突出了肠道失调,细菌转移和BCO之间的联系.
证明了补充益生菌,前生菌和协生菌的潜力,以创造一种优生菌环境并减少BCO.

结论:

肠道微生物组失调有助于肉的BCO.
补充益生菌,益生菌和协生菌提供了一个有希望的策略来缓解BCO.
对生物标志物,病理生理学和宿主-病原体相互作用的进一步研究对于有效的BCO管理至关重要.

关键词:

细菌性冠状腺硬化和骨髓炎 (BCO)生物标志物生物标志物 brojler 肉肉是一个这就是惰,惰,惰.介质细胞是介质细胞.

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