脊髓损伤中的双边副结膜出血:一个病例报告
Junqing Zhong1, Weisheng Ye1, Dan Meng2
1Department of Rehabilitation, Tianjin Hospital, Tianjin, China.
The Journal of international medical research
|August 3, 2023
概括
脊髓损伤患者的神经性膀在抗凝时可能会出现出血并发症. 在尝试通过腹部压力自发排尿之前,仔细评估风险至关重要.
科学领域:
- 神经学 神经学
- 泌尿器科 泌尿器科 泌尿器科 泌尿器科
- 眼科医生 眼科 眼科
背景情况:
- 神经性下尿路功能障碍是脊髓损伤后的常见并发症.
- 脊髓损伤的患者通常需要进行干预,例如尿导管和抗凝治疗.
研究的目的:
- 描述一个患有脊髓损伤和神经性膀的患者双边副结膜出血的病例.
- 为了突出尿导管切除,抗凝和该患者群体出血事件之间的潜在关联.
主要方法:
- 一个45岁男性的病例报告,T12脊髓损伤和神经性膀.
- 监测患者的尿功能,抗凝状态和眼科发现.
- 干预涉及重新插入尿管以控制出血.
主要成果:
- 患者在抗凝治疗期间切除尿导管后出现双边副结膜出血.
- 尿导管重新插入后出血消失.
- 凝血功能和血小板计数处于正常范围内,眼科检查没有显著的结果.
结论:
- 在接受抗凝药治疗的脊髓损伤患者中,神经性膀功能障碍需要仔细评估出血风险.
- 在这种情况下,试图通过腹部压力自发排尿需要评估潜在的出血风险,包括药物审查.
相关概念视频
Hemorrhagic Stroke l: Introduction
A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Hemorrhagic Stroke ll: Pathophysiology
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Secondary Spinal Cord Injury llI: Pathophysiology
Early Ischemia and Ionic ImbalanceWithin minutes of spinal cord injury, a secondary cascade begins, progressing over hours to weeks. Vascular damage reduces blood flow, causing ischemia and mitochondrial dysfunction. ATP depletion leads to ion pump failure, membrane depolarization, sodium influx, potassium efflux, and water accumulation, resulting in cellular swelling. Increased intracellular calcium further disrupts mitochondria and accelerates cellular injury.Excitotoxicity and Neuronal...


