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Updated: Jul 16, 2026

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可以通过MAPK和Nrf2途径在小鼠中改善乙氨基诱导的氧化应激

Mylanayakanahosahalli Chandrashekar Indumathi1, Kamatam Swetha1, Kandahalli Venkataranganayaka Abhilasha2

  • 1Department of Studies in Biochemistry, 8J8C+98P, University of Mysore, Manasagangotri, Mysore, 570006, Karnataka, India.

Biological trace element research
|September 13, 2023
PubMed
概括

补充剂可以保护肝脏免受乙氨基过量毒性的影响. 这项研究表明,可以减少肝损伤生物标志物和氧化应激,为乙氨基诱导的肝毒性提供潜在的治疗策略.

关键词:
乙氨基是一种肝毒性 肝毒性 肝毒性这就是MAPKs.在 Nrf2 和 HO-1 中.氧化应激是一种氧化应激.是一种.

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Mitogen-activated protein kinase, or MAPK pathway, activates three sequential kinases to regulate cellular responses such as proliferation, differentiation, survival, and apoptosis. The canonical MAPK pathway starts with a mitogen or growth factor binding to an RTK. The activated RTKs stimulate Ras, which recruits Raf or MAP3 Kinase (MAPKKK), the first kinase of the MAPK signaling cascade. Raf further phosphorylates and activates MEK or MAP2 Kinases (MAPKK), which in turn phosphorylates MAP...